The research team's next step, in the field of autoimmunity research, involves the design of highly specific small molecules to be tested for their ability to inhibit inflammation and autoimmunity, initially in a mouse model and later in humans.
The ERC Starting Grants are awarded to talented early-career scientists, who have already produced excellent work and show potential to be research leaders, to launch their own projects.
Professor Christos (Kitsos) Louis, IMBB researcher, was honored for his significant contributions to the understanding of the biology of the malaria mosquito on July 27 at the Mosquito Kolymbari Conference 2022, held at the Orthodox Academy of Crete.
Establishment of the Hellenic Network of Molecular Oncology, a Pan-Hellenic hub in the field of oncology
In nature, there is a constant evolutionary battle between microbes and their hosts, during which, many bacteria secrete specific proteins into the host’s cells, aiming to alter its physiology, while increasing pestiferous activity.
A team of scientists from the University of Minnesota Medical School, University of Texas Health San Antonio, and the Biomedical Research Institute of FORTH, found that the disruption of a circadian clock gene may be involved in the development of autism spectrum disorder.
FORTH researchers reveal how metabolic reprogramming in mitochondria promotes or undermines survival and longevity
The new findings of the study are published today in the premier international scientific journal Nature Communications.
How does nature utilize the common repertoire of folds in proteins to differentiate their specificity, and ultimately their function? This long-standing and fundamental question was addressed in a study conducted at the Institute of Molecular Biology and Biotechnology of the Foundation for Research & Technology-Hellas (FORTH-IMBB).
IMBB researchers reveal that DNA damage in tissue-infiltrating macrophages triggers an exosome-based metabolic reprogramming leading to chronic inflammation.
Research carried out at the Institute of Molecular Biology and Biotechnology (IMBB) of FORTH, provides evidence that persistent DNA damage triggers an exosome-based, metabolic reprogramming that leads to chronic inflammation and tissue pathology in DNA repair-deficient progeroid syndromes and likely also during aging.
The findings of the study, published today in the premier international scientific journal Cell Reports, reveal a novel molecular mechanism underlying the detrimental effects of BER imbalance in the pathogenesis of Parkinson’s disease.
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